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A recent investigation revealed that BMSC-Exos can protect against ischemia-reperfusion injury by preventing cardiomyocyte ferroptosis [149]

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At this point, we have previously shown that IGF-I is a main character in restoring mitochondrial dysfunction during aging by increasing mitochondrial membrane potential, reducing oxygen consumption, and increasing ATP synthesis what in turn minimize the c-cytochrome release to the cytoplasm and subsequently promote neural survival by decreasing caspase-induced apoptosis [55,56], in agreement with in vitro reported works from others [242-244]

The additionally calculated stimulation index (SI) of ROS-producing PMN ( Supplementary Table 3 ) showed a biphasic time course with a minimum at 1 h pi and a maximum at 12 h pi